CAUSAS DE LA ENFERMEDAD PULPAR (ETIOLOGÍA)

CONTENIDO DE TODA LA UNIDAD 7 7.1. Inflamación pulpar. 7.1.1. Mecanismos de la inflamación. 7.1.2. Tipos básicos de la inflamación. 7.1.2.1. Reacción inflamatoria aguda. 7.1.2.1.1. Signos macroscópicos de la inflamación. 7.1.2.1.2. Alteraciones vasculares. 7.1.2.1.3. Fagocitosis. 7.1.2.1.4. Exudados y mediadores de la inflamación. 7.1.2.2. Reacción inflamatoria crónica. 7.1.2.2.1. Características. 7.1.2.2.2. Respuesta celular. 7.1.2.2.3. Tejido de granulación. 7.1.3. Efectos remotos de la inflamación. 7.1.4. Regeneración y restauración. 7.1.5. Fisiopatología pulpar. 7.1.5.1. Respuesta pulpar a la caries. 7.2. Etiología de la enfermedad pulpar 7.2.1. Causas físicas. 7.2.2. Causas químicas. 7.2.3. Causas bacterianas. 7.2.4. Caminos de la invasión bacteriana a la pulpa. 7.3. Clasificaciones de las enfermedades pulpares. 7.3.1. Clasificaciones histopatológicas. 7.3.2. Clasificaciones clínicas 7.4. Patología pulpar según Grossman. Sinónimos. Definición. Tipos. Histopatología. Causas. Síntomas. Diagnóstico. Diagnóstico diferencial. Tratamiento Pronostico. de 7.4.1. Pulpitis reversible. 7.4.2. Pulpitis irreversible. 7.4.2.1. Pulpitis aguda. 7.4.2.2. Pulpitis crónica ulcerativa 7.4.2.3. Pulpitis crónica hiperplásica 7.4.2.4. Resorción interna 7.4.5. Degeneración pulpar. 7.4.6. Necrosis pulpar.

PALABRAS CLAVE

Compton Duane Mitchell David “Pharmacologic Treatment of Painful Pulpitis:A Five-Year Report” J Dent Res January-February 1970; 49(1), p.183 (Biblioteca FESI Iztacala)

In 1962 B. F. Lawson and D. F. Mitchell (Oral Surg 17:47-61, 1964) initiated a doubleblind study of a corticoid-antibiotic mixture* as a pulp-capping agent for the treatment of painful pulpitis in permanent teeth. Fifty teeth of 42 patients were treated; half were treated with pure starch and half with 10% erythromycin estolate (Ilosone), 10% streptomycin sulfate, 0.5% fluorandrenolone (Cordran), and qs starch. Each patient had pain, or had recently suffered spontaneous pain, suggestive of pulpitis. Diagnostic procedures and follow-up examinations consisted of the recording of subjective symptoms and the results of objective clinical tests which included percussion, heat, cold, electric vitality, and radiographic tests.

After 92 days, 100% clinical succes s was obtained with the experimental mixture as opposed to 52% with the control starch, with 12 failures. Three teeth were extracted and sectioned for microscopic study after three days, two teeth had been treated with starch and were painful again, the other tooth treated with the corticoid-antibiotic was asymptomatic. Each tooth showed a definite exposure and regions of reparative dentin, which was probably deposited during carious invasion. In the two starch controls, large abscesses were located immediately apical to the treated exposure, but the rest of the pulp chambers were free of inflammation.

Investigado por César Iván López Martínez, Alumno del grupo 2601 (2008), FES Iztacala, UNAM

APOYO ACADÉMICO POR ANTOLOGÍAS

UNIDAD 7: PATOLOGÍA PULPAR

rivasmr@servidor.unam.mx o rivasmr@prodigy.net.mx